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Low Dose Naltrexone


 

The following information is from the low dose naltrexone site www.lowdosenaltrexone.org:

What is low-dose naltrexone and why is it important?

> Low-dose naltrexone holds great promise for the millions of people worldwide with autoimmune diseases or central nervous system disorders or who face a deadly cancer.

Naltrexone itself was approved by the FDA in 1984 in a 50mg dose for the purpose of helping heroin or opium addicts, by blocking the effect of such drugs. By blocking opioid receptors, naltrexone also blocks the reception of the opioid hormones that our brain and adrenal glands produce: beta-endorphin and metenkephalin. Many body tissues have receptors for these endorphins and enkephalins, including virtually every cell of the body's immune system.

 

 

In 1985, Bernard Bihari, MD, a physician with a clinical practice in New York City, discovered the effects of a much smaller dose of naltrexone (approximately 3mg once a day) on the body's immune system. He found that this low dose, taken at bedtime, was able to enhance a patient's response to infection by HIV, the virus that causes AIDS. 

How does LDN work?

 

> LDN boosts the immune system, activating the body's own natural defenses.

The brief blockade of opioid receptors between 2 a.m. and 4 a.m. that is caused by taking LDN at bedtime each night is believed to produce a prolonged up-regulation of vital elements of the immune system by causing an increase in endorphin and enkephalin production. Normal volunteers who have taken LDN in this fashion have been found to have much higher levels of beta-endorphins circulating in their blood in the following days. Animal research by I. Zagon, PhD, and his colleagues has shown a marked increase in metenkephalin levels as well. 

Bihari says that his patients with HIV/AIDS who regularly took LDN before the availability of HAART were generally spared any deterioration of their important helper T cells (CD4+).

In human cancer, research by Zagon over many years has demonstrated inhibition of a number of different human tumors in laboratory studies by using endorphins and low dose naltrexone. It is suggested that the increased endorphin and enkephalin levels, induced by LDN, work directly on the tumors' opioid receptors — and, perhaps, induce cancer cell death (apoptosis). In addition, it is believed that they act to increase natural killer cells and other healthy immune defenses against cancer.

In general, in people with diseases that are partially or largely triggered by a deficiency of endorphins (including cancer and autoimmune diseases), or are accelerated by a deficiency of endorphins (such as HIV/AIDS), restoration of the body's normal production of endorphins is the major therapeutic action of LDN.

LDN has shown beneficial effects many diseases such as:

Cancers:

Bladder Cancer, Breast Cancer, Carcinoid, Colon & Rectal Cancer, Glioblastoma, Liver Cancer Lung Cancer ((Non-Small Cell), Lymphocytic Leukemia (chronic), Lymphoma (Hodgkin's and Non-Hodgkin's), Malignant Melanoma, Multiple Myeloma, Neuroblastoma, Ovarian Cancer, Pancreatic Cancer, Prostate Cancer (untreated), Renal Cell Carcinoma, Throat Cancer, and Uterine Cancer.

Other Diseases:

ALS (Lou Gehrig's Disease), Alzheimer's Disease, Ankylosing Spondylitits, Autism Spectrum Disorders, Behcet's Disease, Celiac Disease, Chronic Fatigue Syndrome, CREST syndrome, Crohn's Disease, Dermatomyositis, Emphysema, Endometriosis, Fibromyalgia, HIV/AIDS, Irritable Bowel Syndrome, Multiple Sclerosis (MS), Myasthenia Gravis (MG), Parkinson's Disease, Pemphigoid, Primary Lateral Sclerosis (PLS), Psoriasis, Rheumatoid Arthritis, Sarcoidosis, Scleroderma, Sjogren's Syndrome, Stiff Person Syndrome, Systemic Lupus (SLE), and Transverse Myelitis.

 

 1. Low Dose Naltrexone. www.lowdosenaltrexone.org. Accessed May 21, 2001.

 

 

 



 

 

 
 
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